Assessment of Renal Erythropoietic Status of the Newly Diagnosed Diabetic Patients without Renal Impairment in Benin City, Nigeria
Grace Umahi-Ottah1, Osehimimhen Precious Inegbedion2, Ojo Moses Oke3, Usman Itakure Abdulkadir4, Nkechi Augustina Olise2, Adewale Adegboyega Oke5, Emmanuel Ojeideleko Akhaumere6, Uche Cletus Odionyenma7, Fidelis Ohiremen Oyakhire8, Iria Kelly Esezobor9, Samson Efenarhua10, Chinemerem Elizabeth Anwara11, Osamudiamen Joshua Eboselume8, Simon Uzor12, Babatunde Ishola Gabriel Adejumo2*orcid
1Department of Physiology, College of Health Sciences, Ebonyi State University, Abakaliki, Ebonyi State, Nigeria.
2Department of Medical Laboratory Science, University of Benin, Benin City, Benin.
3Department of Medical Laboratory Science, College of Health Technology, Akure, Ondo State, Nigeria.
4Department of Medical Laboratory Science, Federal University, Lafia, Nasarawa State, Nigeria.
5Department of Medical Laboratory Science, College of Allied Health Sciences, McPherson University, Ogun State, Nigeria.
6Department of Chemical Pathology, National Hospital, Abuja, Nigeria.
7Department of Medical Laboratory Science, University of Medical Sciences, Ondo, Ondo State, Nigeria.
8Department of Medical Laboratory Science, Faculty of Allied Health Sciences, Benson Idahosa University, Benin City, Edo State, Nigeria.
9Department of Physiology, College of Health Sciences, Joseph Ayo Babalola University, Ikeji-Arakeji, Osun State, Nigeria.
10Department of Natural Science, Faculty of Science and Technology, Middlesex University, London, UK.
11Department of Anatomy, College of Health Sciences, Ebonyi State University, Abakaliki, Ebonyi State, Nigeria.
12Department of Medical Laboratory Science, International Institute for Oncology and Cancer Research, David Umahi Federal University of Health Sciences, Uburu, Ebonyi State, Nigeria.
DOI: 10.4236/health.2024.1610062   PDF    HTML   XML   78 Downloads   508 Views  

Abstract

Diabetes mellitus is a carbohydrate metabolism disorder which is caused due to impairment in insulin secretion and/or the activity of insulin, leading to chronic hyperglycemia with defective carbohydrate, fat and protein metabolism. This study aimed at assessing the erythropoietin (EPO), hemoglobin and renal parameters levels among the newly diagnosed diabetic patients and providing valuable insights into the management and progression of the disease. A case-control study was conducted on samples of 60 consenting participants including newly diagnosed diabetic patients (n − 30), and healthy controls (n − 30) of age ranging between 20 - 50 years. EPO level was measured using enzyme-linked immunosorbent assay (ELISA), the renal parameters (electrolytes) were measured using Ion-Selective Electrodes. Hemoglobin, urea and creatinine were measured using cyanmethemoglobin and colorimetric methods respectively under standard protocols. Demographic and clinical data, including age, gender, diabetes duration, iron rich diet consumption, medication history and family history were collected via questionnaires. Independent sample t-test indicated significantly higher mean hemoglobin (p < 0.05), packed cell volume (p = 0.05) and fasting blood glucose (p < 0.001) in newly diagnosed diabetic patients compared with their healthy control. No significant differences were observed in EPO, creatinine, urea, potassium, bicarbonate, sodium, and chloride between the two groups. In this study, the values of haemoglobin, packed cell volume, EPO and all renal biomarkers were normal, this may be due to the early diagnosis of the disease. It also suggests the extensive capacity of the kidney which is able to withstand metabolic disturbances in the newly diagnosed diabetes mellitus condition. Routine medical check and lifestyle modification are recommended to a newly diagnosed diabetic patients. Also further research is warranted to explore the clinical implications of these assessments in predicting diabetes complications, disease progression and guiding therapeutic interventions.

Share and Cite:

Umahi-Ottah, G. , Inegbedion, O. , Oke, O. , Abdulkadir, U. , Olise, N. , Oke, A. , Akhaumere, E. , Odionyenma, U. , Oyakhire, F. , Esezobor, I. , Efenarhua, S. , Anwara, C. , Eboselume, O. , Uzor, S. and Adejumo, B. (2024) Assessment of Renal Erythropoietic Status of the Newly Diagnosed Diabetic Patients without Renal Impairment in Benin City, Nigeria. Health, 16, 873-887. doi: 10.4236/health.2024.1610062.

1. Introduction

Diabetes mellitus is a carbohydrate metabolism disorder which is caused due to impairment in insulin secretion and/or the activity of insulin, leading to chronic hyperglycemia with defective carbohydrate, fat and protein metabolism [1]. There are two (2) types of diabetes mellitus: Type 1 and Type 2 diabetes mellitus. Type 1 diabetes mellitus is often diagnosed in childhood and this form involves the autoimmune destruction of insulin producing beta cells in the pancreas [2] while type 2 diabetes is the most common and usually diagnosed in adults. It is characterized by insulin resistance and relative insulin deficiency, and it is usually associated with factors like obesity, sedentary lifestyle, and genetic predisposition [3]. Insulin resistance in type 2 diabetes and lack of insulin production in type 1 diabetes leads to chronic hyperglycemia, which disrupts normal metabolic processes, thus, affecting fat, protein and carbohydrate metabolism [4].

Epidemiologically, diabetics is a growing global health issue affecting millions worldwide [5]. In Benin City, the prevalence has been steadily rising, reflecting broader trends in urbanization, lifestyle changes, and genetic predisposition [6]. Several complications such as cardiovascular disease, kidney failure, vision loss and neuropathy [1], result from prolonged diabetes and as such, an early diagnosis is crucial to avoid these complications, reduce morbidity and mortality rate and as well as increasing the patient quality of life. While much attention has been devoted to understanding the conventional markers and biomarkers that may provide valuable insights into the disease pathophysiology and prognosis, one such avenue of investigation involves the assessment of erythropoietin, hemoglobin and renal parameters among newly diagnosed diabetes patients to validate the kidneys efficiency which play a major role in erythropoiesis process.

Erythropoietin, a glycoprotein hormone primarily known for its role in regulating red blood cell production, has demonstrated pleiotropic effect beyond hematopoiesis, including modulation of glucose metabolism and insulin sensitivity [7]. The production is stimulated by hypoxia (low oxygen levels) and is predominantly produced in the kidneys [8]. Long-term high blood sugar levels, a defining characteristic of diabetes, result to complications such as diabetes nephropathy which causes damage of nephrons in the kidney and impairs their function [9]. The interplay of erythropoietin with diabetes involves several mechanisms of action, which includes a reduced capacity to produce erythropoietin, a hormone crucial for stimulating the bone marrow to produce red blood cells, due to damaged kidney seen in diabetes nephropathy [10]. Also, diabetes is often associated with chronic inflammation. The inflammatory cytokines released can interfere with erythropoietin signaling, thereby diminishing its effectiveness in red blood cell production [11]. In addition, chronic hyperglycemia can lead to increased oxidative stress, which may impair the sensitivity of bone marrow to erythropoietin, thus leading to their decreased production [12]. All these factors contribute to the development of anemia in patients with diabetes complications, particularly those with renal involvement [13]. Also, in chronic hyperglycemia, hemoglobin, the oxygen carrying capacity of blood levels are lowered usually due to reduction in production of erythropoietin usually seen in diabetes nephropathy, chronic low-grade inflammation and increased glycation of hemoglobin with other proteins [14].

Diabetic kidney disease, the most serious microangiopathic complication, affects 30% - 40% of people with diabetes. It is responsible for more than 22.6% of cases of chronic end-stage renal disease in France in 2017, its prevalence was 21.6% among dialysis patients in Morocco and 7.8% in Ivory Coast [15]. In Togo, the hospital incidence of rapid renal function decline in diabetic patients is 35% [16]-[18].

Some renal biomarkers such as creatinine, urea and electrolytes levels are interfered in diabetes. Creatinine, a waste product produced by muscle metabolism levels is increased in chronic hyperglycemia due to glomerular damage in complicated diabetes [19]. Also, blood urea nitrogen increases in cases of chronic hyperglycemia, due to impaired glomerular function in complicated diabetes [20]. Electrolyte such as potassium is increased due to reduced excretion of potassium and increased accumulation in the blood [19].

Hyponatremia (low sodium) occurs in chronic hyperglycemia due to osmotic diuresis, where glucose acts as an osmotic agent in the kidneys, pulling water with it into the urine and thus leading to relative loss of sodium [21]. Bicarbonate ion level is reduced in chronic hyperglycemia. The intricate dance of these processes, provides the importance of studying them concurrently, especially in the early stages of diseases, thus allowing for the identification of early biomarkers that may provide valuable insight into predicting disease severity and progression, for the management and treatment of newly diagnosed diabetes patients. To the best of our knowledge, no documented work has been carried out on the assessment of the erythropoiesis status of the newly diagnosed diabetic patients in Benin City. This study therefore tends to assess the erythropoiesis status of the kidney among these group of people.

2. Materials and Method

2.1. Study Design

This is a case control study of patients with newly diagnosed diabetes mellitus who were evaluated for erythropoietin, hemoglobin and some renal parameters levels using their blood sample. A well-structured questionnaire was administered to every participant to obtain basic demographic details as well as anthropometric characteristics. This study involved a cohort of 60 individuals, consisting of 30 newly diagnosed diabetes mellitus patients (both males and females) and 30 deemed healthy controls. The study participants consisted of individuals newly diagnosed of diabetes mellitus attending diabetic clinic University Benin Teaching Hospital Benin city. The control group is made up of healthy people without a history of diabetes mellitus. The inclusion criteria excluded those with severe health complications that made them seemed fragile. Furthermore, those who left the study for any reason were excluded as well. A well-structured consent form was sent after providing a detailed explanation to the participant was in agreement with the researchers and the goal of the study before proceeding further.

2.2. Questionnaire/Ethical Consideration

The questionnaire comprised inquires specifically formulated to obtain information regarding various ages, genders, state of origin, occupation, marital status, family history of diabetes mellitus, underlying disease condition, type of diabetes mellitus, degree/extent of smoking and alcohol consumption, supplement intake, diet consciousness and involvement on physical activity or exercise. Ethical approval with reference number ADME/E 22/A/VOL.VII/14838152172 was obtained from the Health Research Ethics Committee, University of Benin Teaching Hospital, Benin City, Edo State to carry out this study.

2.3. Sample Collection/Preparation

10 millimeters of venous blood from the participants was collected from the participant’s ante-cubital veins using a sterile syringe and needle, and placed into an Ethylene diamine tetra-acetic (EDTA), fluoride oxalate, lithium heparin and plain containers respectively under aseptic conditions. The samples in the ethylenediaminetetra-acetic (EDTA) container were used to examine for hemoglobin and packed cell volume immediately. The samples in the fluoride oxalate container were used to estimate the fasting blood glucose level of the participants. The samples in the lithium heparin container were centrifuged at 5000 rpm for 5 minutes to separate the serum from the clot and used to estimate the electrolyte, urea and creatinine levels. The samples in the plain container were left to clot for a few minutes and centrifuged at 4000 rpm for 5 minutes to separate the serum from the clot. The serum was then dispensed into another clean dry plain container and used to estimate for erythropoietin level of the subjects. All samples were stored at −80˚C prior analysis.

3. Laboratory Investigation

3.1. Determination of Hemoglobin Level

Hemoglobin levels of the participants were determined using cyanmethemoglobin method, according to the manufacturer’s instruction [22].

3.2. Determination of Packed Cell Volume

The Packed Cell Volume was determined using microhematocrit method, according to the manufacturer’s instruction [23].

3.3. Determination of Glucose Level

Glucose levels of the participant was determined using glucose oxidase method while using a Randox glucose reagent with LOT number GAB2002R, and the test was done following the manufacturer’s instructions [24].

3.4. Determination of Urea Level

The urea levels of the participants were determined using colorimetric method, according to the manufacturer’s instructions. The reagent was commercially purchased from Randox company, United Kingdom with LOT number 637861 [25].

3.5. Determination of Creatinine Level

Creatinine level was determined by Jaffe’s method, according to the manufacturer’s instruction [26]. Creatinine reagent was gotten from Randox company with LOT number 630901.

3.6. Determination of Electrolytes Level

Electrolytes levels were determined using ion selective electrode method, using ISE Model 4000; S/N 04020488 from France, according to the manufacturer’s instructions [27].

3.7. Determination of Erythropoietin Level

Erythropoietin level was determined using Elabscience ELISA kit with LOT number ER164VFB1442, according to the manufacturer’s instructions [28].

3.8. Statistical Analysis

Descriptive data were expressed as mean and standard deviation for continuous variables and as percentages for categorical variables. Comparative analysis between two groups was done using independent sample t-test. Association between two continuous variables was done using the Pearson’s bivariate correlation test. Statistical significance was set at p ≤ 0.05. All statistics were performed using SPSS for windows (version 25.0).

4. Results

Table 1 shows the socio-demographic characteristics of the study population. The study population comprises 60 participants (newly diagnosed diabetics, n = 30, and healthy controls, n = 30) of age ranging between 20 - 50 years (mean ± SD, 37.17 ± 6.24 years). A greater percentage of the participants were males (54.1%), <40 years (67.2%), married (59%), employed (57.4%), had tertiary education (95.1%) and from Bini kingdom (39.3%).

Some selected life-styles of the study population are shown in Table 2. Data shows that a greater percentage of the healthy control (90.3%) and patients living with newly diagnosed with diabetes (90%) do not smoke. Majority of the control (67.7%) and patients newly diagnosed with diabetes (73.3%) do not drink alcohol. Majority of the participants (control, 29.0%); patients living with newly diagnosed diabetes, (33.3%) stated that they eat diet containing iron. All the control reported that they are not on regulated food diet, while all the diabetic patients said their diets were regulated. Most of the participants (control, 51.6%; newly diagnosed diabetics, 63.3%) reported that they engage in moderate exercise.

Table 1. Socio-demographic characteristics of the participants.

Characteristics

Frequency

Percent (%)

Sex

Females

28

45.9

Males

32

54.1

Age group

<40 years

41

67.2

≥40 years

19

32.8

Ethnicity

Akoko Edo

7

11.5

Bini

24

39.3

Esan

15

26.2

Etsako

6

9.8

Igbo

7

11.5

Urobo

1

1.6

Marital Status

Divorced

2

3.3

Married

35

59.0

Single

23

37.7

Occupation

Employed

35

57.4

Retired

4

6.6

Unemployed

21

36.1

Educational Status

Primary

3

4.9

Tertiary

57

95.1

Table 2. Selected life-style characteristics of the study population.

Lifestyle Variables

Groups

Total

Control

(n = 30)

Newly Diagnosed Diabetics

(n = 30)

Smoking

No

28 (90.3%)

27 (90.0%)

55 (90.2%)

Yes

2 (9.7%)

3 (10.0%)

6 (9.8%)

Alcohol

No

21 (67.7%)

22 (73.3%)

43 (70.5%)

Yes

9 (32.3%)

8 (26.7%)

18 (29.5%)

Iron Diet

Frequently

9 (29.0%)

10 (33.3%)

19 (31.1%)

Occasionally

13 (41.9%)

11 (36.7%)

24 (39.4%)

Rarely

8 (29.0%)

9 (30.0%)

18 (29.5%)

Regulated Food

No

30 (100%)

0 (0%)

31 (50.8%)

Yes

0 (0%)

30 (100%)

30 (49.2%)

Exercise

Lightly

11 (35.5%)

9 (30.0%)

20 (32.8%)

Moderate

15 (51.6%)

19 (63.3%)

35 (57.4%)

Sedentary

0 (0%)

1 (3.3%)

1 (1.6%)

Vigorous

4 (12.9%)

1 (3.3%)

5 (8.2%)

The clinical characteristics of the patients living with newly diagnosed diabetes mellitus is shown in Table 3. Overall diabetes patients’ data indicated that majority (70%) of the patients were suffering from type 2 diabetes. A greater percentage (53.3%) of the patients had a family history of diabetes. Most of the patients (86.7%) had suffered diabetes for a period of 1 to 6 months. As at the time of the study, 73.3% of the patients had no underlying disease conditions. However, ten percent of the patients were suffering from frequent dehydration and 6.7% were suffering from ulcer. None of the patients was reported taking diabetes medications.

Table 4 shows the mean levels of erythropoietin, hemoglobin, packed cell volume and renal parameters in patients with newly diagnosed diabetes mellitus and their healthy control. Independent sample t-test indicated significantly higher mean hemoglobin (p < 0.05), packed cell volume (p = 0.05), and fasting blood glucose (p < 0.001) in newly diagnosed diabetic patients compared with their healthy control. In contrast, no significant differences were observed in erythropoietin, urea, creatinine, potassium, bicarbonate, sodium, chloride between the two groups.

Table 5 shows the relationship among the renal biomarkers in healthy control group. Peardon’s bivariate correlation test indicated no significant relationships between erythropoietin and other parameters of renal function. Hemoglobin indicated significant positive correlations with Hb (p < 0.001), urea (p = 0.009), and creatinine (p < 0.001), but not with K+, HCO 3 , Na+, Cl and FBG. Packed cell volume indicated significant positive correlations with urea (p = 0.008), and creatinine, but not K+, HCO 3 , Na+, Cl and FBG. Urea was positively associated with creatinine (p = 0.003) and potassium (p = 0.040), but not with HCO 3 , Na+, Cl and FBG. Creatinine was positively associated with bicarbonate (p = 0.045), but not with K+, Na+, Cl and FBG. Potassium was positively associated with bicarbonate (p < 0.001), chloride (p = 0.010) and fasting blood glucose (p = 0.022), but not with Na+. Bicarbonate indicated significant correlation with fasting blood glucose (p = 0.005) but not with Na+, and Cl. Sodium indicated positive correlation with chloride (p = 0.004), but not with FBG. There was no significant relationship between Chloride and FBG.

Table 3. Clinical characteristics of patients living with newly diagnosed diabetes mellitus.

Characteristics

Frequency

Percent (%)

Type of Diabetes Mellitus

Type 1

9

30.0

Type 2

21

70.0

Family History

No

14

46.7

Yes

16

53.3

Duration of Diabetes Mellitus

1 - 6 Months

26

86.7

7 - 12 Months

4

13.3

Underlying Disease

None

22

73.3

Congenital Issues

1

3.3

Excessive Sweating

1

3.3

Frequent Dehydration

3

10.0

Joint pain

1

3.3

Ulcer

2

6.7

Medication

No

30

100.0

Table 4. Mean levels of erythropoietin, hemoglobin, packed cell volume and renal parameters in patients with newly diagnosed diabetes mellitus and their healthy control.

Variables

Groups

N

Mean

Std. Deviation

T-Statistics

p-Value

EPO (IU/L)

Control

30

6.95

1.06

0.20

0.837

Newly Diagnosed Diabetics

30

6.90

1.00

HB (g/dl)

Control

30

14.59

1.87

−2.26

0.027

Newly Diagnosed Diabetics

30

15.84

2.39

PCV (%)

Control

30

43.91

5.50

−1.97

0.05

Newly Diagnosed Diabetics

30

47.38

8.00

Urea (mg/dl)

Control

30

25.25

3.60

1.51

0.15

Newly Diagnosed Diabetics

30

29.44

8.53

Creatinine (mg/dl)

Control

30

0.67

0.08

−1.17

0.245

Newly Diagnosed Diabetics

30

1.53

4.05

Potassium (mmol/L)

Control

30

4.06

0.38

1.09

0.280

Newly Diagnosed Diabetics

30

3.92

0.62

Bicarbonate (mmol/L)

Control

30

21.64

1.79

0.09

0.925

Newly Diagnosed Diabetics

30

21.57

4.01

Sodium (mmol/L)

Control

30

135.97

2.07

0.91

0.364

Newly Diagnosed Diabetics

30

132.46

21.19

Chloride (mmol/L)

Control

30

101.64

2.69

−0.30

0.762

Newly Diagnosed Diabetics

30

102.06

7.20

FBG (mg/dl)

Control

30

87.32

13.41

−9.48

<0.001

Newly Diagnosed Diabetics

30

177.83

51.36

Table 5. Relationship among the renal biomarkers (erythropoietin, urea, creatinine, and electrolytes levels), hemoglobin, PCV and FBG in healthy control group.

EPO

HB

PCV

UREA

Cr

K+

HCO 3

Na+

Cl

FBG

EPO

R

1

p

HB

R

0.195

1

p

0.294

PCV

R

0.189

0.998**

1

p

0.308

<0.001

Urea

R

0.171

0.460**

0.469**

1

p

0.356

0.009

0.008

Cr

R

0.088

0.669**

0.662**

0.512**

1

p

0.637

<0.001

<0.001

0.003

K+

R

0.031

0.085

0.072

0.371*

0.256

1

p

0.869

0.650

0.699

0.040

0.164

HCO 3

R

0.176

0.087

0.077

0.210

0.362*

0.606**

1

p

0.344

0.642

0.680

0.257

0.045

<0.001

Na+

R

0.010

0.336

0.339

0.113

0.262

0.172

0.131

1

p

0.958

0.065

0.062

0.546

0.154

0.355

0.483

Cl

R

0.029

0.067

0.070

0.243

0.213

0.454*

0.228

0.506**

1

p

0.876

0.719

0.709

0.187

0.250

0.010

0.217

0.004

FBG

R

0.056

-0.104

-0.117

0.124

0.110

0.411*

0.495**

-0.107

0.248

1

p

0.763

0.576

0.531

0.505

0.557

0.022

0.005

0.565

0.179

Abbreviations: EPO, Erythropoietin; Hb, hemoglobin, PCV, Packed Cell Volume; Cr, Creatinine; K+ Potassium; HCO 3 , Bicarbonate; Na+, Sodium; Cl, Chloride; FBG, Fasting Blood Sugar; R, Correlation coefficient, p, Significant value. **. Correlation is significant at the 0.01 level; *. Correlation is significant at the 0.05 level.

5. Discussion

Diabetes, also referred to as diabetes mellitus, is a collection of common endocrine disorders characterized by persistently elevated levels of glucose in the bloodstream [29] [30]. Erythropoietin (EPO) is a glycoprotein classified as a cytokine, a signaling molecule involved in intercellular communication [31]. Primarily produced and secreted by specialized cells within the kidneys, EPO plays a vital role in erythropoiesis, the physiological process of red blood cell production. The production of EPO is tightly regulated by oxygen homeostasis. When oxygen levels in tissues fall below a certain threshold, hypoxia-inducible factors (HIFs) are activated within the kidney [32]. Therefore, EPO serves as a critical regulator of red blood cell homeostasis, ensuring adequate oxygen delivery to tissues throughout the body.

Recent studies suggest a potential link between erythropoietin (EPO) levels, kidney function, and newly diagnosed diabetes. Hyperglycemia, a hallmark of diabetes, and chronic inflammation may suppress EPO production in the kidneys, a key organ for EPO synthesis. This could contribute to anemia, frequently observed in diabetics. Moreover, reduced kidney function, measured by GFR, might further decrease EPO, creating a vicious cycle.

In this study, the greater proportion of newly diagnosed uncomplicated diabetic patients are of age ranging between 20 - 50 years. This is in accordance with Bai et al. (2021) [33], that the advancement of diabetes to complicated diabetes increased with advanced age. And also, a study conducted by Sosale et al. (2014) [34], reported that majority of newly diagnosed patients with T2D are from the age group of ≤50.

Also in this study, the selected life-style characteristics of the study population is shown in Table 2, it revealed that all the healthy non-diabetic control reported that they eat non-regulated food diet, while all the diabetic patients said their diets were regulated, while both groups reported that they engage in moderate exercise. This is in agreement with a study by Zhang et al. (2018) [35], that calorie restriction and a focus on nutrient-dense foods, such as fruits, vegetables, and whole grains, have been demonstrated to effectively lower blood sugar levels by preventing postprandial hyperglycemia and that regular physical activity enhances insulin sensitivity through mechanisms involving increased skeletal muscle glucose uptake and improved insulin signaling.

In this study, the clinical characteristics of newly diagnosed diabetic patients was shown in Table 3, which shows that 70% of the patients were suffering from Type 2 diabetes. This result is in agreement with a study conducted by Sosale et al. (2014) [34], which also reported a high prevalence of T2DM among newly diagnosed diabetic patients. And this study also reported a high prevalence of diabetes in individuals with a family history of the disease, which is also in agreement with a study conducted by Annis et al. (2005) [36], which reported that family history of diabetes was a significant predictor of self-reported diabetes among diabetic patients. And that adults with a family history of diabetes in a parent or sibling had four times the odds of having diabetes than adults without a family history of the disease. The study also reported that Family history of type 2 diabetes is recognized as an important risk factor of the disease. Individuals who have a family history of diabetes can have two to six times the risk of type 2 diabetes compared with individuals with no family history of type 2 diabetes. Of the thirty 30 (100%) newly diagnosed diabetic patients in this study, 16 (53.3%) have family history of the disease, while 14 (46.7%) do not have.

In this study, the erythropoietin mean value was observed to be non-statistically significant, this is in agreement with a study conducted by Volker and Radko, (2018) [37], that the kidney complications observed in diabetes mellitus is dependent on how advanced the disease is and how long the patient have been diagnosed of the disease. There are many factors that can stimulate the production of EPO. They include smoking, iron level and exercise. In this study, 3 (10%) of the thirty newly diagnosed diabetes patients do smoke, while 27 (90%) stated in the questionnaire that they do not smoke. Also, of the total number, 10 (33.3%) frequently take iron rich diet, 11 (36.7%) occasionally do take, while 9 (30%) rarely take iron rich diet. 9 (30%) of the newly diagnosed diabetes mellitus indulged in light exercise, 19 (63.3%) moderate, while 1 (3.3%) represent those involved in vigorous exercise and sedentary life respectively.

Also, the result shows that there is higher mean value of hemoglobin (p < 0.05), packed cell volume (p = 0.05), and fasting blood glucose (p < 0.001) in newly diagnosed diabetic patients compared with their healthy control. In contrast, no significant differences were observed in urea, creatinine, potassium, bicarbonate, sodium, chloride between the two groups. The high mean value of fasting blood glucose of 177.83 mg/dl reported in this study (Table 4), is in agreement with a study conducted by Nahar et al. (2011) [38], which also reported a high value of fasting blood glucose among newly diagnosed type 2 diabetes patients.

In this study, greater percentage of the newly diagnosed diabetic patients had a high mean value of hemoglobin which is also in agreement with a study by Lee et al. (2018) [39], their study reported a high value of hemoglobin among type 2 diabetic patients when compared with the healthy non-diabetic group and also in this study, it was reported that the study participants were on iron rich diets which is an important requirement needed by the body for the production of hemoglobin. It is established that the value of hemoglobin and PCV are inter-related and are directly proportional, as an increase in hemoglobin will also lead to increase in PCV value, hence the increase in PCV value in this study. The high level of hemoglobin and packed cell volume may be due to fact that majority of the patients consumed iron rich diet, which is necessary for the erythropoiesis. All the 30 (100%) newly diagnosed patients admitted to be involved in lifestyle modifications such as moderate physical exercise which is shown to improve blood circulation, and oxygen delivery to tissues, which can stimulate the production of red blood cells and increase hemoglobin levels overtime. Exercise is known to stimulate the production of EPO which in turn stimulates the production of red blood cell.

In this study, it was also observed that there were no significant differences in mean creatinine level between the control group and newly diagnosed diabetic patients. This is in disagreement with a study conducted by Schneider et al. (2016) [40] which reported that high creatinine level in the blood is associated with diabetic nephropathy, which is mostly seen in advance cases of diabetes or in complicated diabetes. It was also shown that the urea and electrolytes level were not statistically significant. The reason for the above results may be due to the fact that the patients in this study were diagnosed on time, as none of them has shown any signs of diabetic derangement at the point of diagnosis. Early diabetes mainly affects how the body uses blood sugar for energy, with no direct impact on kidneys. However, uncontrolled diabetes can lead to high blood sugar levels damaging the kidneys in later stages.

6. Conclusion

In this study, the values of hemoglobin, packed cell volume, EPO and all renal biomarkers were normal, this may be due to the early diagnosis of the disease. It also suggests the extensive capacity of the kidney which is able to withstand metabolic disturbances in the newly diagnosed diabetes mellitus condition. Routine medical check, including the assessment of renal status and lifestyle modification are recommended to a newly diagnosed diabetic patients, and all their offspring and family members. Also, further research is warranted to explore the clinical implications of these assessments in predicting diabetes complications, disease progression and guiding therapeutic interventions.

Acknowledgement

We acknowledge the ethical committee of the University of Benin Teaching Hospital, Benin City, Edo State, and all the participants.

Funding

The research was privately funded.

Conflicts of Interest

The authors declare no conflicts of interest regarding the publication of this paper.

References

[1] American Diabetes Association (2009) Diagnosis and Classification of Diabetes Mellitus. Diabetes Care, 32, S62-S67.
https://doi.org/10.2337/dc09-s062
[2] Atkinson, M.A., Eisenbarth, G.S. and Michels, A.W. (2014) Type 1 Diabetes. The Lancet, 383, 69-82.
https://doi.org/10.1016/s0140-6736(13)60591-7
[3] Hu, F.B. (2011) Globalization of Diabetes. Diabetes Care, 34, 1249-1257.
https://doi.org/10.2337/dc11-0442
[4] DeFronzo, R.A. (2009) From the Triumvirate to the Ominous Octet: A New Paradigm for the Treatment of Type 2 Diabetes Mellitus. Diabetes, 58, 773-795.
https://doi.org/10.2337/db09-9028
[5] International Diabetes Federation (2019) IDF Diabetes Atlas. 9th Edition, International Diabetes Federation.
[6] Unnikrishnan, R., Pradeepa, R., Joshi, S.R. and Mohan, V. (2020) Type 2 Diabetes: Demystifying the Global Epidemic. Journal of Diabetes, 69, 1054-1066.
[7] Mikolás, E., Cseh, J., Pap, M., Szijárto, I., Balogh, A., Laczy, B., et al. (2012) Effects of Erythropoietin on Glucose Metabolism. Hormone and Metabolic Research, 44, 279-285.
https://doi.org/10.1055/s-0032-1301901
[8] Jelkmann, W. (2013) Physiology and Pharmacology of Erythropoietin. Transfusion Medicine and Hemotherapy, 40, 302-309.
https://doi.org/10.1159/000356193
[9] Alicic, R.Z., Rooney, M.T. and Tuttle, K.R. (2017) Diabetic Kidney Disease. Clinical Journal of the American Society of Nephrology, 12, 2032-2045.
https://doi.org/10.2215/cjn.11491116
[10] Mehdi, U. and Toto, R.D. (2009) Anemia, Diabetes, and Chronic Kidney Disease. Diabetes Care, 32, 1320-1326.
https://doi.org/10.2337/dc08-0779
[11] Gluba-Brzózka, A., Franczyk, B., Olszewski, R. and Rysz, J. (2020) The Influence of Inflammation on Anemia in CKD Patients. International Journal of Molecular Sciences, 21, Article 725.
https://doi.org/10.3390/ijms21030725
[12] Maiese, K., Chong, Z., Hou, J. and Shang, Y. (2008) Erythropoietin and Oxidative Stress. Current Neurovascular Research, 5, 125-142.
https://doi.org/10.2174/156720208784310231
[13] Thomas, M.C. (2006) The Role of Anemia in Progression of Diabetic Kidney Disease. Journal of the American Society of Nephrology, 17, S19-S21.
[14] Williams, A., Bissinger, R., Shamaa, H., Patel, S., Bourne, L., Artunc, F., et al. (2023) Pathophysiology of Red Blood Cell Dysfunction in Diabetes and Its Complications. Pathophysiology, 30, 327-345.
https://doi.org/10.3390/pathophysiology30030026
[15] Gallagher, H. and Suckling, R.J. (2016) Diabetic Nephropathy: Where Are We on the Journey from Pathophysiology to Treatment? Diabetes, Obesity and Metabolism, 18, 641-647.
https://doi.org/10.1111/dom.12630
[16] Tsevi, Y., Amekoudi, E.Y.M. and Sabi, T.S.E.V.I. (2021) Déclin rapide de la fonction rénale chez les patients diabétiques à Lomé (Togo). Néphrologie & Thérapeutique, 17, 310-311.
https://doi.org/10.1016/j.nephro.2021.07.166
[17] Kossi, K., Mawufemo, T.Y., Badomta, D., Georges, T.K., Abago, B., Hubert, Y.K., et al. (2022) Renal Risk among Diabetic Patients in Togo. Open Journal of Nephrology, 12, 249-261.
https://doi.org/10.4236/ojneph.2022.123026
[18] Mohamud, A.M., Xu, N., Liu, G., Odilov, B., Jiang, B. and Hu, Z. (2022) The Spectrum of Kidney Disease in Type Two Diabetic Patients: A Single-Center Study. Open Journal of Nephrology, 12, 1-14.
https://doi.org/10.4236/ojneph.2022.121001
[19] Liamis, G. (2014) Diabetes Mellitus and Electrolyte Disorders. World Journal of Clinical Cases, 2, 488-496.
https://doi.org/10.12998/wjcc.v2.i10.488
[20] Zhong, J., Yao, Y., Zeng, G., Zhang, Y., Ye, B., Dou, X., et al. (2023) A Closer Association between Blood Urea Nitrogen and the Probability of Diabetic Retinopathy in Patients with Shorter Type 2 Diabetes Duration. Scientific Reports, 13, Article No. 9881.
https://doi.org/10.1038/s41598-023-35653-z
[21] Huang, Y., Liu, W., Liu, J., Guo, D., Zhang, P., Liu, D., et al. (2021) Association of Urinary Sodium Excretion and Diabetic Kidney Disease in Patients with Type 2 Diabetes Mellitus: A Cross-Sectional Study. Frontiers in Endocrinology, 12, Article 772073.
https://doi.org/10.3389/fendo.2021.772073
[22] Nkrumah, B., Nguah, S.B., Sarpong, N., Dekker, D., Idriss, A., May, J., et al. (2011) Hemoglobin Estimation by the Hemocue Portable Hemoglobin Photometer in a Resource Poor Setting. BMC Clinical Pathology, 11, Article No. 5.
https://doi.org/10.1186/1472-6890-11-5
[23] Farooq, U., Idris, M., Sajjad, N., Lashari, M.H., Ahmad, S., Rehman, Z.U., et al. (2023) Investigating the Potential of Packed Cell Volume for Deducing Hemoglobin: Cholistani Camels in Perspective. PLOS ONE, 18, e0280659.
https://doi.org/10.1371/journal.pone.0280659
[24] Kumar, V. and Gill, K.D. (2018) Estimation of Blood Glucose Levels by Glucose Oxidase Method. In: Kumar, V. and Gill, K.D., Eds., Basic Concepts in Clinical Biochemistry: A Practical Guide, Springer, 57-60.
https://doi.org/10.1007/978-981-10-8186-6_13
[25] Langenfeld, N.J., Payne, L.E. and Bugbee, B. (2021) Colorimetric Determination of Urea Using Diacetyl Monoxime with Strong Acids. PLOS ONE, 16, e0259760.
https://doi.org/10.1371/journal.pone.0259760
[26] Toora, B.D. and Rajagopal, G. (2002) Measurement of Creatinine by Jaffe’s Reaction-Determination of Concentration of Sodium Hydroxide Required for Maximum Color Development in Standard, Urine and Protein Free Filtrate of Serum. Indian Journal of Experimental Biology, 40, 352-354.
[27] Rayana, M.C.B., Burnett, R.W., Covington, A.K., D'Orazio, P., Fogh-Andersen, N., Jacobs, E., et al. (2006) Recommendation for Measuring and Reporting Chloride by Ises in Undiluted Serum, Plasma or Blood: International Federation of Clinical Chemistry and Laboratory Medicine (IFCC): IFCC Scientific Division, Committee on Point of Care Testing and Working Group on Selective Electrodes. Clinical Chemistry and Laboratory Medicine, 44, 346-352.
https://doi.org/10.1515/cclm.2006.060
[28] Prabhakar, S.P. and Ganesan, M. (2015) Development of a High Sensitive Sandwich ELISA for Measuring Erythropoietin in Human Serum. Applied Biological Research, 17, 105-112.
https://doi.org/10.5958/0974-4517.2015.00018.x
[29] Schoener, B. and Borger, J. (2024) Erythropoietin Stimulating Agents. StatPearls Publishing.
https://www.ncbi.nlm.nih.gov/books/NBK536997/
[30] Haase, V.H. (2010) Hypoxic Regulation of Erythropoiesis and Iron Metabolism. American Journal of Physiology-Renal Physiology, 299, F1-F13.
https://doi.org/10.1152/ajprenal.00174.2010
[31] Galicia-Garcia, U., Jebari, S., Larrea-Sebal, A., Uribe, K.B., Siddiqi, H., Ostolaza, H., et al. (2020) Statin Treatment-Induced Development of Type 2 Diabetes: From Clinical Evidence to Mechanistic Insights. International Journal of Molecular Sciences, 21, Article 4725.
https://doi.org/10.3390/ijms21134725
[32] Yadav, U.N., Ghimire, S., Mistry, S.K., Shanmuganathan, S., Rawal, L.B. and Harris, M. (2021) Prevalence of Non-Communicable Chronic Conditions, Multimorbidity and Its Correlates among Older Adults in Rural Nepal: A Cross-Sectional Study. BMJ Open, 11, e041728.
https://doi.org/10.1136/bmjopen-2020-041728
[33] Bai, A., Tao, J., Tao, L. and Liu, J. (2021) Prevalence and Risk Factors of Diabetes among Adults Aged 45 Years or Older in China: A National Cross-Sectional Study. Endocrinology, Diabetes & Metabolism, 4, e00265.
https://doi.org/10.1002/edm2.265
[34] Sosale, A., Prasanna Kumar, K., Sadikot, S., Nigam, A., Bajaj, S., Zargar, A., et al. (2014) Chronic Complications in Newly Diagnosed Patients with Type 2 Diabetes Mellitus in India. Indian Journal of Endocrinology and Metabolism, 18, 355-360.
https://doi.org/10.4103/2230-8210.131184
[35] Zhang, J., Yang, Z., Xiao, J., Xing, X., Lu, J. and Chen, L. (2017) Association between Insulin Resistance and Gestational Diabetes Mellitus and Risk of Preeclampsia: A Meta-Analysis. Medicine, 96, e7041.
[36] Annis, A.M., Caulder, M.S., Cook, M.L. and Duquette, D. (2005) Family History, Diabetes, and Other Demographic and Risk Factors Among Participants of the National Health and Nutrition Examination Survey 1999–2002. Prevalence of Chronic Disease, 2, A19.
[37] Volker, V. and Radko, K. (2018) Pathophysiology of the Diabetic Kidney. Journal of Comprehensive Physiology, 1, 1175-1232.
[38] Nahar, S., Rahman, M., Ullah, M., Debnath, B., Sultana, N. and Farhad, C. (1970) Prevalence of Metabolic Syndrome in Newly Diagnosed Type 2 Diabetes Mellitus. Cardiovascular Journal, 4, 17-25.
https://doi.org/10.3329/cardio.v4i1.9385
[39] Lee, M., Han, K., Lee, J., Sohn, S., Jeong, J., Kim, M., et al. (2018) High Hemoglobin Levels Are Associated with Decreased Risk of Diabetic Retinopathy in Korean Type 2 Diabetes. Scientific Reports, 8, Article No. 5538.
https://doi.org/10.1038/s41598-018-23905-2
[40] Meier, C., Schneider, C., Jick, S. and Coll, B. (2016) Doubling of Serum Creatinine and the Risk of Cardiovascular Outcomes in Patients with Chronic Kidney Disease and Type 2 Diabetes Mellitus: A Cohort Study. Clinical Epidemiology, 8, 177-184.
https://doi.org/10.2147/clep.s107060

Copyright © 2025 by authors and Scientific Research Publishing Inc.

Creative Commons License

This work and the related PDF file are licensed under a Creative Commons Attribution 4.0 International License.