Cheng-Xue, R., Gómez-Ruiz, A., Antoine, N., Noël, L.A., Chae, H.-Y., Ravier, M.A., Chimienti, F., Schuit, F.C. and Gilon, P. (2013) Tolbutamide Controls Glucagon Release from Mouse Islets Differently than Glucose. Involvement of KATP Channels from Both α-Cells and α-Cells. Diabetes, 62, 1612-1622.
http://dx.doi.org/10.2337/db12-0347

has been cited by the following article:

• TITLE: Role of Calcium in Glucagon Secretion

  AUTHORS: Willy J. Malaisse

  KEYWORDS: Calcium Ions, Glucagon Secretion, Verapamil, Nutrional Factors, Microtubular-Microfilamentous System

  JOURNAL NAME: Advances in Biological Chemistry, Vol.4 No.3, April 22, 2014

  ABSTRACT: The role of calcium ions in the process by which nutrients affect glucagon secretion by pancreatic islet α-cells remains the matter of an apparently endless debate. In the prolongation of recent articles dealing with this matter, the present review draws attention to the dual role of Ca2+ as revealed by prior publications. In such a perspective, emphasis is placed on the increase in glucagon output in response to the omission of extracellular Ca2+ as recorded in the presence of D-glucose or 2-ketoisocaproate, the permissive role of extracellular Ca2+ in the positive secretory response to arginine or a mixture of fumarate, glutamate and pyruvate, and the effects of an organic calcium-antagonist on glucagon output. Considering the role currently ascribed to Ca2+ in the activation of motile events involved in stimulus-secretion coupling, attention is also given to the effects of cytochalasin B, D2O and mitotic-spindle inhibitors upon the secretory response of α-cells exposed to D-glucose in the absence or presence of arginine.