has been cited by the following article(s):
[1]
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Two Small Molecules, ZCL278 and AZA197 Show Promise in Influencing Protein Interactions Involving the Ras-Related Protein Cell division cycle 42 [Cdc42] to Modulate Its Oncogenic Potential
Open Journal of Biophysics,
2017
DOI:10.4236/ojbiphy.2017.73006
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[2]
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A ‘synthetic-sickness’ screen for senescence re-engagement targets in mutant cancer backgrounds
PLOS Genetics,
2017
DOI:10.1371/journal.pgen.1006942
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[3]
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Intrinsic GTP hydrolysis is observed for a switch 1 variant of Cdc42 in the presence of a specific GTPase inhibitor
Small GTPases,
2016
DOI:10.1080/21541248.2015.1123797
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[4]
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The value of genomics in dissecting the RAS-network and in guiding therapeutics for RAS-driven cancers
Seminars in Cell & Developmental Biology,
2016
DOI:10.1016/j.semcdb.2016.06.012
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[5]
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Direct Attack on RAS: Intramolecular Communication and Mutation-Specific Effects
Clinical Cancer Research,
2015
DOI:10.1158/1078-0432.CCR-14-2148
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