"Macrophage phenotypes correspond with remodeling outcomes of various acellular dermal matrices"
written by Hitesh Agrawal, Sunil S. Tholpady, Anthony E. Capito, David B. Drake, Adam J. Katz,
published by Open Journal of Regenerative Medicine, Vol.1 No.3, 2012
has been cited by the following article(s):
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[1] A comparison of patient-reported outcomes between Alloderm and Dermacell in immediate alloplastic breast reconstruction: A randomized control trial
[2] Outcome of split thickness skin grafts on excised burns with different dermal compositions
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[4] Designing a Novel Immunomodulatory Surface Modification to Promote Biomaterial-Tissue Integration
[5] Allograft Tissue Safety and Technology
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[7] Using Immunofluorescent Cell Marker Quantification as an Identification Method for M1 and M2 Macrophage Phenotypes
[8] Long‐term follow‐up comparison of two different bi‐layer dermal substitutes in tissue regeneration: Clinical outcomes and histological findings
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[9] A randomized clinical trial of a human acellular dermal matrix demonstrated superior healing rates for chronic diabetic foot ulcers over conventional care and an active …
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[10] Evaluation of Tumour Associated Macrophages and Angiogenesis in Ameloblastoma
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[11] Utilization of a Fetal Bovine Xenograft (PriMatrix) to Achieve Dental Root Coverage: A Pilot Study
[12] The host response to naturally-derived extracellular matrix biomaterials
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[14] Unbiased Analysis of the Impact of Micropatterned Biomaterials on Macrophage Behavior Provides Insights beyond Predefined Polarization States
ACS Biomaterials Science & Engineering, 2017
[15] Image based Machine Learning for identification of macrophage subsets
[16] Can host reaction animal models be used to predict and modulate skin regeneration?
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[17] The impact of surface chemistry modification on macrophage polarisation
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[18] Asynchronous inflammation and myogenic cell migration limit muscle tissue regeneration mediated by acellular scaffolds
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[19] Electrophoretically decellularized xenogeneic extracellular matrix for large volume skeletal muscle regeneration
[20] Sequential delivery of immunomodulatory cytokines to facilitate the M1-to-M2 transition of macrophages and enhance vascularizati
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[21] Macrophages Modulate Engineered Human Tissues for Enhanced Vascularization and Healing
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[22] Transplantation of devitalized muscle scaffolds is insufficient for appreciable de novo muscle fiber regeneration after volumetr
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[23] The role of macrophage phenotype in vascularization of tissue engineering scaffolds
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[24] Asynchronous inflammation and myogenic cell migration limit muscle tissue regeneration mediated by a cellular scaffolds
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