When a Glue Sniffer Turns Weak ()
Abstract
Toluene inhalation can result in
electrolyte and acid-base derangements and should be considered in the
differential diagnosis of young patients with unexplained hypokalaemia and normal anion gap metabolic acidosis. This case serves to
illustrate the abnormalities and heighten awareness among emergency physicians
who may not have laboratory results on hand when evaluating causes of limb weakness.
Share and Cite:
Juliana, P. and Seth, P. (2012) When a Glue Sniffer Turns Weak.
International Journal of Clinical Medicine,
3, 769-771. doi:
10.4236/ijcm.2012.37A135.
Conflicts of Interest
The authors declare no conflicts of interest.
References
[1]
|
D. C. Batlle, S. Sabatini and N. A. Kurtzman, “On the Mechanism of Toluene-Induced Renal Tubular Acidosis,” Nephron, Vol. 49, No. 3, 1998, pp. 210-218.
doi:10.1159/000185057
|
[2]
|
E. J. F. Carlisle, S. M. Donnelly, et al., “Glue-Sniffing and Distal Renal Tubular Acidosis: Sticking to the Facts,” Journal of the American Society of Nephrology, Vol. 1, No. 8, 1991, pp. 1019-1027.
|
[3]
|
J.-J Hong, J.-L Lin, et al., “Chronic Glue Sniffer with Hy perchloraemia Metabolic Acidosis, Rhabdomyolysis, Irreversible Quadriplegia, Central Pontine Myelinolysis and Hypothyroidism,” Nephrology Dialysis Transplant, Vol. 11, No. 9, 1996, pp. 1848-1849.
doi:10.1093/oxfordjournals.ndt.a027683
|
[4]
|
Richard H. Bennett, Harvey R. Forman, “Hypokalemic Periodic Paralysis in Chronic Toluene Exposure,” Archives of Neurology, Vol. 37, No. 10, 1980, p. 673.
doi:10.1001/archneur.1980.00500590097021
|