Neuroscience and Medicine

Volume 2, Issue 3 (September 2011)

ISSN Print: 2158-2912   ISSN Online: 2158-2947

Google-based Impact Factor: 0.67  Citations  

The Unexpected Capability of Melanin to Split the Water Molecule and the Alzheimer’s Disease

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DOI: 10.4236/nm.2011.23029    11,500 Downloads   24,551 Views  Citations

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ABSTRACT

We began a study about the three main causes of blindness in 1990, because their incidence and prevalence have not changed in the last forty years. Twelve years later we concluded that the main source of energy for the human retina is water, not ATP. And this is also true for the entire human body. Water is the main source of energy. The amazing capability of eukaryotic cells to break or dissociate the water molecule was unsuspected to us because it takes 2000℃ degrees to dissociate water in a laboratory environment, and until today, it was believed that only plants were capable of accomplishing this. Photosynthesis occurs in humans as it does in plants. The water that we drink every day is not just to wash away detritus and toxins; it is not just a cleaner, nor a simple solvent. When our body dissociates the water molecule, cells are able to get their energy from Hydrogen (Hydrogen is the energy carrier that Nature uses the most). Water is our main source of energy. If our body couldn’t acquire energy from water, we would need to eat between 50 - 170 kg (110 - 374 lbs) daily. In any system, when a generalized failure occurs, we must suspect energy first. Parkinson and Alzheimer’s Disease are examples of a generalized failure. That explains why it is not uncommon that patients improve dramatically with pharmacological stimulation of the human photosynthesis process. Recall that the brain needs energy not only to grow or to perform its functions, but also to preserve its form and shape. The best energy for human cells is Hydrogen.

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M. Arias-Esparza, R. Arias, P. Arias, M. Arias and A. Solís-Herrera, "The Unexpected Capability of Melanin to Split the Water Molecule and the Alzheimer’s Disease," Neuroscience and Medicine, Vol. 2 No. 3, 2011, pp. 217-221. doi: 10.4236/nm.2011.23029.

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