[1]
|
P. H. Schiller, J. H. Sandell and J. H. Maunsell, “Functions of the ON and OFF Channels of the Visual System,” Nature, Vol. 322, No. 6082, 1986, pp. 824-825.
doi:10.1038/322824a0
|
[2]
|
E. V. Famiglietti and H. Kolb, “Structural Basis for On- Center and Off-Center Responses in Retinal Ganglion- Cells,” Science, Vol. 194, No. 4261, 1976, pp. 193-195.
doi:10.1126/science.959847
|
[3]
|
R. Nelson, E. V. Famiglietti and H. Kolb, “Intracellular Staining Reveals Different Levels of Stratification for On-Center and Off-Center Ganglion-Cells in Cat Retina,” Journal of Neurophysiology, Vol. 41, No. 2, 1978, pp. 472-483.
|
[4]
|
J. Ammermuller and H. Kolb, “The Organization of the Turtle Inner Retina. 1. On-Center and Off-Center Pathways,” Journal of Comparative Neurology, Vol. 358, 1995, pp. 1-34. doi:10.1002/cne.903580102
|
[5]
|
S. H. Devries, “Selective Distribution of AMPA and Kainate Receptors at the Mammalian Cone to OFF Bipolar Cell Synapse,” Investigative Ophthalmology & Visual Science, Vol. 41, 2000, p. S621.
|
[6]
|
E. J. Chichilnisky and R. S. Kalmar, “Functional Asymmetries in ON and OFF Ganglion cells of Primate Retina,” Journal of Neuroscience, Vol. 22, No. 7, 2002, pp. 2737-2747.
|
[7]
|
B. B. Lee, “Receptive Field Structure in the Primate Retina,” Vision Research, Vol. 36, No. 5, 1996, pp. 631-644.
doi:10.1016/0042-6989(95)00167-0
|
[8]
|
W. H. Merigan and J. H. R. Maunsell, “How Parallel Are the Primate Visual Pathways,” Annual Review of Neuroscience, Vol. 16, 1993, pp. 369-402.
doi:10.1146/annurev.neuro.16.1.369
|
[9]
|
J. Pokorny and V. C. Smith, “Psychophysical Signatures Associated with Magnocellular and Parvocellular Pathway Contrast Gain,” Journal of the Optical Society of America A: Optics Image Science and Vision, Vol. 14, No. 9, 1997, pp. 2477-2486. doi:10.1364/JOSAA.14.002477
|
[10]
|
S. Plainis and I. J. Murray, “Neurophysiological Interpretation of Human Visual Reaction Times: Effect of Contrast, Spatial Frequency and Luminance,” Neuropsychologia, Vol. 38, No. 12, 2000, pp. 1555-1564.
doi:10.1016/S0028-3932(00)00100-7
|
[11]
|
C. Wehrhahn and D. Rapf, “ON- and OFF-Pathways form Separate Neural Substrates for Motion Perception: Psychophysical Evidence,” Journal of Neuroscience, Vol. 12, No. 6, 1992, pp. 2247-2250.
|
[12]
|
C. Wehrhahn and D. Rapf, “Perceptual Learning of Apparent Motion Mediated through ON- and OFF-Pathways in Human Vision,” Vision Research, Vol. 41, No. 3, 2001, pp. 353-358.
doi:10.1016/S0042-6989(00)00232-7
|
[13]
|
P. H. Schiller, “The ON and OFF Channels of the Visual- System,” Trends in Neurosciences, Vol. 15, No. 3, 1992, pp. 86-92. doi:10.1016/0166-2236(92)90017-3
|
[14]
|
R. W. Bowen, J. Pokorny and V. C. Smith, “Sawtooth Contrast Sensitivity: Decrements Have the Edge,” Vision Research, Vol. 29, No. 11, 1989, pp. 1501-1509.
doi:10.1016/0042-6989(89)90134-X
|
[15]
|
R. W. Bowen, J. Pokorny, V. C. Smith and M. A. Fowler, “Sawtooth Contrast Sensitivity: Effects of Mean Illuminance and Low Temporal Frequencies,” Vision Research, Vol. 32, No. 7, 1992, pp. 1239-1247.
doi:10.1016/0042-6989(92)90218-8
|
[16]
|
K. A. Zaghloul, K. Boahen and J. B. Demb, “Different Circuits for ON and OFF Retinal Ganglion Cells Cause Different Contrast Sensitivities,” Journal of Neuroscience, Vol. 23, No. 7, 2003, pp. 2645-2654.
|
[17]
|
R. S. Anderson and C. OBrien, “Psychophysical Evidence for a Selective Loss of M Ganglion Cells in Glaucoma,” Vision Research, Vol. 37, No. 8, 1997, pp. 1079-1083.
doi:10.1016/S0042-6989(96)00260-X
|
[18]
|
K. R. Alexander, C. S. Barnes, G. A. Fishman, J. Pokorny and V. C. Smith, “Contrast Sensitivity Deficits in Inferred Magnocellular and Parvocellular Pathways in Retinitis Pigmentosa,” Investigative Ophthalmology & Visual Science, Vol. 45, 2004, pp. 4510-4519.
doi:10.1167/iovs.04-0188
|
[19]
|
E. A. Ansari, J. E. Morgan and R. J. Snowden, “Psychophysical Characterisation of Early Functional Loss in Glaucoma and Ocular Hypertension,” British Journal of Ophthalmology, Vol. 86, No. 10, 2002, pp. 1131-1135.
doi:10.1136/bjo.86.10.1131
|
[20]
|
H. Demirci, A. Gezer, F. Sezen, T. Ovali, T. Demiralp and U. Isoglu-Alkoc, “Evaluation of the Functions of the Parvocellular and Magnocellular Pathways Strabismic Amblyopia,” Journal of Pediatric Ophthalmology & Strabismus, Vol. 39, No. 4, 2002, pp. 215-221.
|
[21]
|
A. M. McKendrick, D. K. Badcock and W. H. Morgan, “Psychophysical Measurement of Neural Adaptation Abnormalities in Magnocellular and Parvocellular Pathways in Glaucoma,” Investigative Ophthalmology & Visual Science, Vol. 45, No. 6, 2004, pp. 1846-1853.
doi:10.1167/iovs.03-1225
|
[22]
|
Y. Shan, M. L. Moster, R. A. Roemer and J. B. Siegfried, “Abnormal Function of the Parvocellular Visual System in Anisometropic Amblyopia,” Journal of Pediatric Ophthalmology & Strabismus, Vol. 37, 2000, pp. 73-78.
|
[23]
|
K. R. Alexander, J. Pokorny, V. C. Smith, G. A. Fishman and C. S. Barnes, “Contrast Discrimination Deficits in Retinitis Pigmentosa Are Greater for Stimuli That Favor the Magnocellular Pathway,” Vision Research, Vol. 41, No. 5, 2001, pp. 671-683.
doi:10.1016/S0042-6989(00)00286-8
|
[24]
|
K. R. Alexander, C. S. Barnes, G. A. Fishman and A. H. Milam, “Nature of the Cone ON-Pathway Dysfunction in Melanoma-Associated Retinopathy,” Investigative Ophthalmology & Visual Science, Vol. 43, No. 4, 2002, pp. 1189-1197.
|
[25]
|
J. B. Demb, G. M. Boynton, M. Best and D. J. Heeger, “Psychophysical Evidence for a Magnocellular Pathway Deficit in Dyslexia,” Vision Research, Vol. 38, No. 11, 1998, pp. 1555-1559.
doi:10.1016/S0042-6989(98)00075-3
|
[26]
|
K. R. Alexander, C. S. Barnes and G. A. Fishman, “ON-Pathway Dysfunction and Timing Properties of the Flicker ERG in Carriers of X-Linked Retinitis Pigmentosa,” Investigative Ophthalmology & Visual Science, Vol. 44, No. 9, 2003, pp. 4017-4025.
doi:10.1167/iovs.02-0989
|
[27]
|
C. S. Barnes, K. R. Alexander and G. A. Fishman, “A Distinctive form of Congenital Stationary Night Blindness with Cone ON-Pathway Dysfunction,” Ophthalmology, Vol. 109, 2002, pp. 575-583.
doi:10.1016/S0161-6420(01)00981-2
|
[28]
|
J. E. Wolf and G. B. Arden, “Selective Magnocellular Damage in Melanoma-Associated Retinopathy: Comparison with Congenital Stationary Nightblindness,” Vision Research, Vol. 36, No. 15, 1996, pp. 2369-2379.
doi:10.1016/0042-6989(95)00316-9
|
[29]
|
K. Benoff, K. Fitzgerald, V. Zemon, E. Pinkhasov, J. Gordon and G. Cibis, “Magnocellular ON-Pathway Deficits in Duchenne Muscular Dystrophy: A Visual Evoked Potential Study,” Investigative Ophthalmology & Visual Science, Vol. 42, 2001, p. S787.
|
[30]
|
A. M. McKendrick and D. R. Badcock, “Constrast-Processing Dysfunction in Both Magnocellular and Parvocellular Pathways in Migraineurs with or without Aura,” Investigative Ophthalmology & Visual Science, Vol. 44, 2003, pp. 442-448. doi:10.1167/iovs.02-0630
|
[31]
|
A. I. Klistorner and S. L. Graham, “Early Magnocellular Loss in Glaucoma Demonstrated Using the Pseudorandomly Stimulated Flash Visual Evoked Potential,” Journal of Glaucoma, Vol. 8, No. 2, 1999, pp. 140-148.
doi:10.1097/00061198-199904000-00010
|
[32]
|
A. M. McKendrick, G. P. Sampson, M. J. Walland and D. R. Badcock, “Contrast Sensitivity Changes due to Glaucoma and Normal Aging: Low-Spatial-Frequency Losses in both Magnocellular and Parvocellular Pathways,” Investigative Ophthalmology & Visual Science, Vol. 48, No. 5, 2007, pp. 2115-2122. doi:10.1167/iovs.06-1208
|
[33]
|
H. Sun, W. H. Swanson, B. Arvidson and M. W. Dul, “Assessment of Contrast Gain Signature in Inferred Magnocellular and Parvocellular Pathways in Patients with Glaucoma,” Vision Research, Vol. 48, No. 26, 2008, pp. 2633-2641. doi:10.1016/j.visres.2008.04.008
|
[34]
|
A. R. Davis, J. J. Sloper, M. M. Neveu, C. R. Hogg, M. J. Morgan and G. E. Holder, “Differential Changes of Magnocellular and Parvocellular Visual Function in Early- and Late-Onset Strabismic Amblyopia,” Investigative Ophthalmology & Visual Science, Vol. 47, No. 11, 2006, pp. 4836-4841. doi:10.1167/iovs.06-0382
|
[35]
|
K. R. Alexander, A. S. Rajagopalan, W. Seiple, V. M. Zemon and G. A. Fishman, “Contrast Response Properties of Magnocellular and Parvocellular Pathways in Retinitis Pigmentosa Assessed by the Visual Evoked Potential,” Investigative Ophthalmology & Visual Science, Vol. 46, No. 8, 2005, pp. 2967-2973.
doi:10.1167/iovs.05-0231
|
[36]
|
P. D. Butler, I. Schechter, L. A. DeSanti, J. Maddox, J. M. Harkavy-Friedman, V. C. Greenstein, et al., “Examination of Magnocellular and Parvocellular Visual Pathway Function in Schizophrenia Using Electrophysiological and Psychophysical Techniques,” Schizophrenia Research, Vol. 36, 1999, p. 161.
|
[37]
|
P. D. Butler, I. Schechter, V. Zemon, S. G. Schwartz, V. C. Greenstein, J. Gordon, et al., “Dysfunction of Early-Stage Visual Processing in Schizophrenia,” American Journal of Psychiatry, Vol. 158, No. 7, 2001, pp. 1126-1133. doi:10.1176/appi.ajp.158.7.1126
|
[38]
|
A. K. Ludlow, A. J. Wilkins and P. Heaton, “The Effect of Coloured Overlays on Reading Ability in Children with Autism,” Journal of Autism and Developmental Disorders, Vol. 36, No. 8, 2006, pp. 507-516.
doi:10.1007/s10803-006-0090-5
|
[39]
|
E. Pellicano, L. Gibson, M. Maybery, K. Durkin and D. R. Badcock, “Abnormal Global Processing along the Dorsal Visual Pathway in Autism: A Possible Mechanism for Weak Visuospatial Coherence?” Neuropsychologia, Vol. 43, No. 7, 2005, pp. 1044-1053.
doi:10.1016/j.neuropsychologia.2004.10.003
|
[40]
|
N. J. Rinehart, J. L. Bradshaw, A. V. Brereton and B. J. Tonge, “A Clinical and Neurobehavioural Review of High-Functioning Autism and Asperger’s Disorder,” Australian and New Zealand Journal of Psychiatry, Vol. 36, No. 6, 2002, pp. 762-770.
|
[41]
|
R. Hecker and B. Mapperson, “Dissociation of Visual and Spatial Processing in Working Memory,” Neuropsychologia, Vol. 35, No. 5, 1997, pp. 599-603.
doi:10.1016/S0028-3932(96)00106-6
|
[42]
|
D. B. Bednarek and A. Grabowska, “Luminance and Chromatic Contrast Sensitivity in Dyslexia: The Magnocellular Deficit Hypothesis Revisited,” Neuroreport, Vol. 13, No. 18, 2002, pp. 2521-2525.
doi:10.1097/00001756-200212200-00028
|
[43]
|
B. J. W. Evans, N. Drasdo and I. L. Richards, “Dyslexia: The Link with Visual Deficits,” Ophthalmic and Physiological Optics, Vol. 16, No. 1, 1996, pp. 3-10.
doi:10.1016/0275-5408(95)00021-6
|
[44]
|
D. Omtzigt, A. W. Hendriks and H. H. J. Kolk, “Evidence for Magnocellular Involvement in the Identification of Flanked Letters,” Neuropsychologia, Vol. 40, No. 12, 2002, pp. 1881-1890.
doi:10.1016/S0028-3932(02)00069-6
|
[45]
|
G. W. Stuart, K. I. McAnally and A. Castles, “Can Contrast Sensitivity Functions in Dyslexia Be Explained by Inattention Rather Than a Magnocellular Deficit?” Vision Research, Vol. 41, No. 24, 2001, pp. 3205-3211.
doi:10.1016/S0042-6989(01)00176-6
|
[46]
|
P. H. Schiller, “The ON and OFF Channels of the Mammalian Visual System,” Progress in Retinal and Eye Research, Vol. 15, 1995, pp. 173-195.
doi:10.1016/1350-9462(95)00009-7
|