Hoffman, D.J., Eagles-Smith, C.A., Ackerman, J.T., Adelsbach, T.L. and Stebbins, K.R. (2011) Oxidative stress response of Forster’s terns (Sterna forsteri) and Caspian terns (Hydroprogne caspia) to mercury and selenium bioaccumulation in liver, kidney, and brain. Environmental Toxicology & Chemistry, 30, 920-929.doi10.1016/j.bbamcr.2012.01.005 - References

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Hoffman, D.J., Eagles-Smith, C.A., Ackerman, J.T., Adelsbach, T.L. and Stebbins, K.R. (2011) Oxidative stress response of Forster’s terns (Sterna forsteri) and Caspian terns (Hydroprogne caspia) to mercury and selenium bioaccumulation in liver, kidney, and brain. Environmental Toxicology & Chemistry, 30, 920-929. doi:10.1016/j.bbamcr.2012.01.005

has been cited by the following article:

TITLE: The activities of endothelial elastase and cathepsin G in rats at oxidative stress caused by heavy metals salts

AUTHORS: Nickolaj F. Starodub, Ljubov M. Samokhina

KEYWORDS: Endothelial Elastase; Cathepsin G; Cobalt Chloride; Mercury Chloride; Rats

JOURNAL NAME: Advances in Biological Chemistry, Vol.3 No.2, April 30, 2013

ABSTRACT: CoCl2 introduction increased cathepsin G activity in the heart and liver as well as endothelial elastase (EEl) in kidney that indicated the development of destructive processes. CoCl2 introduction decreased EEl and cathepsin G activities in blood serum and cathepsin G in lungs. HgCl2 injection decreased EEl in blood serum, heart, liver, kidney and cathepsin G in blood serum. These decreasing of proteinases activities may be caused by cytotoxic effects of heavy metals and/or the inclusion of these proteases in the destructive processes and absence of their synthesis and/or release.

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