Mahmoud, Z., Ladislas, R., Dominique, C., Pierre-Jean, T., Latifa, B. and Pierre, D. (2002) Serum elastase activity, serum elastase inhibitors, and occurrence of carotid atherosclerotic plaques. Circulation, 105, 2638-2645. doi10.1161/01.CIR.0000017329.51160.EF - References

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Mahmoud, Z., Ladislas, R., Dominique, C., Pierre-Jean, T., Latifa, B. and Pierre, D. (2002) Serum elastase activity, serum elastase inhibitors, and occurrence of carotid atherosclerotic plaques. Circulation, 105, 2638-2645. doi:10.1161/01.CIR.0000017329.51160.EF

has been cited by the following article:

TITLE: The activities of endothelial elastase and cathepsin G in rats at oxidative stress caused by heavy metals salts

AUTHORS: Nickolaj F. Starodub, Ljubov M. Samokhina

KEYWORDS: Endothelial Elastase; Cathepsin G; Cobalt Chloride; Mercury Chloride; Rats

JOURNAL NAME: Advances in Biological Chemistry, Vol.3 No.2, April 30, 2013

ABSTRACT: CoCl2 introduction increased cathepsin G activity in the heart and liver as well as endothelial elastase (EEl) in kidney that indicated the development of destructive processes. CoCl2 introduction decreased EEl and cathepsin G activities in blood serum and cathepsin G in lungs. HgCl2 injection decreased EEl in blood serum, heart, liver, kidney and cathepsin G in blood serum. These decreasing of proteinases activities may be caused by cytotoxic effects of heavy metals and/or the inclusion of these proteases in the destructive processes and absence of their synthesis and/or release.

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