Article citationsMore>>
van Doorslaer, K., zur Hausen, H., Villiers, E.M., Bernard, H.U., Chen, Z. and Burk, R.S. (2011) Papillomaviruses: Evolution, Linnaean Taxonomy and Current Nomenclature. Trends in Microbiology, 19, 49-50.
http://dx.doi.org/10.1016/j.tim.2010.11.004
has been cited by the following article:
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TITLE:
Prevention of Iatrogenic Cervical Cancer
AUTHORS:
Rudolf Klimek
KEYWORDS:
Cervical Cancer, Cancer Cell, HPV
JOURNAL NAME:
Open Journal of Obstetrics and Gynecology,
Vol.5 No.14,
November
30,
2015
ABSTRACT: Cervical carcinogenesis consists of natural occurring spontaneous
cellular processes which may lead to self-organized dissipative structures of
cervical cancers what was first explained in 1977 after several years of my
biochemical, biophysical, hormonal and clinical studies. That was possible
thanks to monograph “Biochemie der Tumoren” written in 1942 by Nobel Prize
winner H. von Euler with my master Prof. B. Skarzynski. Today I express my
gratitude to Nobelist Harald zur Hausen and his team for they discovered the
nuclide sequences of HPV in genomes of cervical cancer cells which opened the
possibility to describe the causal role of information in formula of reality.
Vaccines built from the protein capsid of HPV have proved only the pathogenic
information about the virus because of its lack of DNAs. All the theories of
carcinogenesis have properly described this event from methodologically
different point of view. The point is that one should understand the
thermodynamic rules underlying each of these approaches. Neoplasms are self-organized
from the cells of the patient, who did not provide the necessary conditions for
cellular metabolism as defined in the moment of appearance of its zygote. In
light of medical thermodynamics all oncogenic factors can divide into
sufficient or necessary to events for creating a dissipathogenic cellular
status. Cervical cancer is a tumor associated with the human papillomavirus as
only its pathogenic dissipathogenic factors, but the genome of cervical
carcinoma cells maybe the original source of many types of HPV from the peeled
off cancer cells of the uterine cervix. Many things are known to increase the
risk of carcinogenesis which as a natural process is an alternative of cellular
or social death. Neoplasm cell is an effect of carcinogenesis, but not a causal
point at which it begins its existence.
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