Laurén, J., Gimbel, D.A., Nygaard, H., Gilbert, J.W. and Strittmatter, S.M. (2009) Cellular Prion Protein Mediates Impairment of Synaptic Plasticity by Amyloid-Beta Oligomers. Nature, 457, 1128-1132. - References

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Laurén, J., Gimbel, D.A., Nygaard, H., Gilbert, J.W. and Strittmatter, S.M. (2009) Cellular Prion Protein Mediates Impairment of Synaptic Plasticity by Amyloid-Beta Oligomers. Nature, 457, 1128-1132. http://dx.doi.org/10.1038/nature07761

has been cited by the following article:

TITLE: Mapping It out: A Novel Signaling Pathway Linking Aβ-PrPc-Fyn Complex to Cognitive Impairment in Alzheimer’s Disease

AUTHORS: Yiling Wang, Lei Xue

KEYWORDS: Alzheimer’s; PrPc; Aβ; Fyn; Tau; Microtubule

JOURNAL NAME: Advances in Alzheimer's Disease, Vol.3 No.1, March 12, 2014

ABSTRACT: Understanding the signaling cascade that leads to the rapid memory and cognitive breakdown in Alzheimer’s disease is the key to finding a potential treatment method for the disease. Recently, Larson et al. connect the roles of major proteins implicated in the disease progression and propose targeting of cellular prion protein (PrPc) as a way of someday rescuing synaptic plasticity in humans with Alzheimer’s, as it has been done on mice.

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