has been cited by the following article(s):
[1]
|
Blood-brain barrier leakage in Alzheimer’s disease: From discovery to clinical relevance
Pharmacology & Therapeutics,
2022
DOI:10.1016/j.pharmthera.2022.108119
|
|
|
[2]
|
Proinflammatory cytokines induce accumulation of glypican-1-derived heparan sulfate and the C-terminal fragment of β-cleaved APP in autophagosomes of dividing neuronal cells
Glycobiology,
2020
DOI:10.1093/glycob/cwaa011
|
|
|
[3]
|
The cyanobacterial neurotoxin β-N-methylamino-l-alanine prevents addition of heparan sulfate to glypican-1 and increases processing of amyloid precursor protein in dividing neuronal cells
Experimental Cell Research,
2019
DOI:10.1016/j.yexcr.2019.03.041
|
|
|
[4]
|
Mouse models of Alzheimer’s disease cause rarefaction of pial collaterals and increased severity of ischemic stroke
Angiogenesis,
2018
DOI:10.1007/s10456-018-9655-0
|
|
|
[5]
|
A protocol for quantitative analysis of murine and human amyloid-β 1-40 and 1-42
Journal of Neuroscience Methods,
2017
DOI:10.1016/j.jneumeth.2017.07.022
|
|
|
[6]
|
Hypoxia induces NO-dependent release of heparan sulfate in fibroblasts from the Alzheimer mouse Tg2576 by activation of nitrite reduction
Glycobiology,
2016
DOI:10.1093/glycob/cww007
|
|
|