Advances in Alzheimer's Disease

Advances in Alzheimer's Disease

ISSN Print: 2169-2459
ISSN Online: 2169-2467
www.scirp.org/journal/aad
E-mail: aad@scirp.org
"Lipolysaccharide-Induced Neuroinflammation Is Associated with Alzheimer-Like Amyloidogenic Axonal Pathology and Dendritic Degeneration in Rats"
written by Xiaohua Deng, Meili Li, Weiming Ai, Lixin He, Dahua Lu, Peter R. Patrylo, Huaibin Cai, Xuegang Luo, Zhiyuan Li, Xiao-Xin Yan,
published by Advances in Alzheimer's Disease, Vol.3 No.2, 2014
has been cited by the following article(s):
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[17] Gram-negative bacteria and their lipopolysaccharides in Alzheimer's disease: pathologic roles and therapeutic implications
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[18] Complete spatial characterisation of N-glycosylation upon striatal neuroinflammation in the rodent brain
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[21] 6‐Gingerol, an active constituent of ginger, attenuates lipopolysaccharide‐induced oxidation, inflammation, cognitive deficits, neuroplasticity, and amyloidogenesis in …
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[24] Inhibition of CD38 and supplementation of nicotinamide riboside ameliorate lipopolysaccharide-induced neuroinflammation in the hippocampus
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[26] Ipriflavone and Ipriflavone loaded albumin nanoparticles reverse lipopolysaccharide induced neuroinflammation in rats
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[27] Orally Administered Antibiotics Vancomycin and Ampicillin Cause Cognitive Impairment With Gut Dysbiosis in Mice With Transient Global Forebrain Ischemia
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[28] Chronic administration of a positive allosteric modulator at the α5-GABAA receptor reverses age-related dendritic shrinkage
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[31] Protein arginine methyltransferase-1 deficiency restrains depression-like behavior of mice by inhibiting inflammation and oxidative stress via Nrf-2
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[32] Geraniin Attenuates Lipopolysaccharide-Induced Cognitive Impairment in Mice by Inhibiting Toll-Like Receptor 4 Activation
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[33] Phytomedicine-Based Potent Antioxidant, Fisetin Protects CNS-Insult LPS-Induced Oxidative Stress-Mediated Neurodegeneration and Memory Impairment
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[34] CB2 Cannabinoid receptor agonist ameliorates novel object recognition but not spatial memory in transgenic APP/PS1 mice
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[36] Lipopolysaccharide-induced neuroinflammation as a bridge to understand neurodegeneration
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[37] Gut Microbiota: From the Forgotten Organ to a Potential Key Player in the Pathology of Alzheimer's Disease
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[38] Inhibitory effect of INT-777 on lipopolysaccharide-induced cognitive impairment, neuroinflammation, apoptosis, and synaptic dysfunction in mice
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[39] The small molecule CA140 inhibits the neuroinflammatory response in wild-type mice and a mouse model of AD
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[46] Probable mechanisms involved in the antipsychotic-like activity of methyl jasmonate in mice
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[48] Glucose tolerance and insulin sensitivity are impaired in APP/PS1 transgenic mice prior to amyloid plaque pathogenesis and cognitive decline
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[49] Methyl jasmonate ameliorates memory deficits in mice exposed to passive avoidance paradigm
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[50] Sortilin fragments deposit at senile plaques in human cerebrum
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[51] Aggregation of thrombin-derived C-terminal fragments as a previously undisclosed host defense mechanism
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[52] Can brain impermeable BACE1 inhibitors serve as anti-CAA medicine?
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