Hirata, T., Fujioka, M., Takahashi, K.A., Arai, Y., Asano, T., Ishida, M., Kuribayashi, M., Akioka, K., Okamoto, M., Yoshimura, N., Satomi, Y., Nishino, H., Fukushima, W., Hirota, Y., Nakajima, S., Kato, S. and Kubo, T. (2007) ApoB C7623T Polymorphism Predicts Risk for Steroid-Induced Osteonecrosis of the Femoral Head after Renal Transplantation. Journal of Orthopaedic Science, 12, 199-206.
http://dx.doi.org/10.1007/s00776-007-1110-9

has been cited by the following article:

• TITLE: Effect of Qing’e Pill plus Salvia on Non-Traumatic Osteonecrosis of the Femoral Head of Idiopathic Type in Earlier Stage: A Case Report of a Twelve-Month of Period

  AUTHORS: Bo Shuai, Yanping Yang, Lin Shen, Manxiang Wu

  KEYWORDS: Qing’e Pill, Osteonecrosis, Femoral Head, Ischemic, Biomarker, Traditional Chinese Medicine

  JOURNAL NAME: Chinese Medicine, Vol.5 No.2, June 19, 2014

  ABSTRACT: Background: Compromised circulatory and bone/lipid metabolic dysfunction are two major contributors to non-traumatic osteonecrosis of the femoral head (ONFH). Qing’e pill plus Salvia has significant anti-inflammatory and anti-atherosclerotic action, and it could also regulate bone formation and remodeling by suppressing osteoclasts. Case Report: We describe a case of a 62-year- old man with ONFH of idiopathic type in earlier stage that could not be adequately controlled with oral non-steroidal analgesics and restoring bone loss agents for past 3 years. After six months treatment with Qing’e pill plus Salvia, the plasma low-density lipoprotein (LDL), high-density lipoprotein (HDL), apolipoprotein A1 (apo A1), apolipoprotein B (apo B), total cholesterol (TC), triglycerides (TG), and blood viscosity were measured on an empty stomach. The Harris scores were maintained during a six months follow-up after discontinuation of Qing’e pill plus Salvia. Discussion: Our case has shown that plasma levels of inflammatory and atherosclerotic relaxed factors and the Harris scores may be controlled with herbal medication in ONFH of idiopathic type in earlier stage that could not be adequately controlled with oral non-steroidal analgesics and restoring bone loss agents.