Browse Menu >>

Browse Subjects >>

Biomedical & Life Sciences

Business & Economics

Chemistry & Materials Science

Computer Science & Communications

Earth & Environmental Sciences

Engineering

Medicine & Healthcare

Physics & Mathematics

Social Sciences & Humanities

Why Us? >>

  • - Open Access
  • - Peer-reviewed
  • - Rapid publication
  • - Lifetime hosting
  • - Free indexing service
  • - Free promotion service
  • - More citations
  • - Search engine friendly

Free SCIRP Newsletters>>

Add your e-mail address to receive free newsletters from SCIRP.

 

Contact Us >>

WhatsApp  +86 18163351462(WhatsApp)
   
Paper Publishing WeChat
Book Publishing WeChat
(or Email:book@scirp.org)

Article citations

More>>

Miller, W.S. (1950) The Lung. 2nd Edition, Charles C Thomas Publishers, Springfield.

has been cited by the following article:

  • TITLE: Entropy Change of Lungs: Determinant of the Static Properties of the Lungs

    AUTHORS: Kyongyob Min

    KEYWORDS: Energy Elasticity, Entropy-Change Considerations, Elastic Properties of Lungs, Thermodynamics

    JOURNAL NAME: Applied Mathematics, Vol.6 No.8, July 8, 2015

    ABSTRACT: The static properties of the lungs have been explained by energy-change considerations on the elasticity, but this article explains the elasticity of the lungs by entropy-change considerations. Entropy of the individual lobule was defined by application of stochastic geometry on aggregated alveolar polyhedrons. Entropy of the lungs is the result of integrating a number of lobular entropies through the fractal bronchial tree. Entropy of the lungs was thus determined by the individual lobular entropy and the connectivity of the bronchial tree to the lobular bronchioles. Thermody-namic considerations on the static conditions of the pulmonary system composed of the lungs and the chest wall have provided a theoretical approach to understand the subdivisions of lung volume as the entropy-change of lungs. Entropy-change considerations on the elasticity of the lungs have shown that alveolar collapse and subsequent alveolar induration as the primary pathway for the loss of elasticity in the lungs is an acceptable hypothesis.