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A Novel Model of Cancer

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DOI: 10.4236/jct.2018.95034    311 Downloads   634 Views  
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ABSTRACT

This study describes a novel model of cancer. Cancer is caused, according to this model, by two toxic molecules, COF2 (carbonyl fluoride) and CS2 (carbon disulfide). COF2 acts as a catalyst and produces CS2. CS2 acts as a catalyst and produces COF2. Hence, the molecules COF2 and CS2 have the ability to reproduce each other, i.e. to create a chain reaction where COF2 and CS2 are multiplied. COF2 and CS2 are toxic. It is proposed that long-term cell exposure to COF2 and CS2 may disturb cell apoptosis and cause uncontrolled cell growth, i.e. cancer. Consequently, cancer is caused by a disease causing mechanism or agent and not by a pathogen. However, cancer can be initiated by e.g. a bacteria or virus that contains the molecules COF2 and CS2, wherein these molecules start a chain reaction producing COF2 and CS2 in the infected area and where the result can be cancer. The study describes why Fe2O3 (iron (III) oxide) may have a therapeutic effect on cancer.

1. Introduction

The present report describes a novel model of cancer. According to this model cancer is caused by a mechanism consisting of two toxic molecules. It is proposed that they impair the immune system. A new method is described that facilitates non-invasive analyses of these two molecules in cancer cell lines and tumors in vivo. These molecules create catalyzes, also in a test tube positioned outside the cell line or tumor. The result is analyzed and provides a description of the mechanism causing cancer.

Within medicine enzyme catalyzes is the increase in the rate of a chemical reaction by the active site of a protein. However, the human organism, pathogens, molecules and chemical elements encompass a much larger range of different types of catalyzes. Catalyzes can create new molecules and where the catalyst is another molecule, compound or chemical element [1] . Molecules and chemical elements can act as catalysts where the catalytic reaction is reduction (destruction) of molecules [2] . Catalyzes is caused by radiated energy from molecules and chemical elements and generates reactions also at distance, similar to the electrons field [3] .

Recent development displays that many pathogens (e.g. bacteria, virus) as well as autoimmune diseases contain a generic part that consists of the molecules COF2 and CS2. This will be reported in separate studies.

An overwhelming body of evidence displays that relationship among certain bacteria and cancer exists [4] . Chronic gastritis induced by Helibacter pylori is the strongest known risk factor for gastric cancer [5] . An estimated 15% of all human cancers worldwide may be attributed to viruses [6] [7] . Several studies have shown that a history of autoimmune diseases increases the risk of cancer [8] , despite the fact that cancer and autoimmune diseases are fundamentally different pathological conditions [9] .

Numerous reports display that the molecule Fe2O3 increases cellular necrosis, increases the rate of apoptosis [10] and significantly inhibits cellular growth [11] . Fe2O3 is the active substance in Ferumoxytol [12] . Ferumoxytol is an FDA-approved therapeutic for the treatment of anemia. However, reports display that it also may be effective in treatment of various forms of cancer [13] [14] .

2. Novel Model of Cancer

Cancer is caused, according to this model, by two molecules COF2 and CS2. Presence of COF2 and CS2 in cancer cell lines and tumors/metastases in vivo can be measured by their radiated frequency spectrum (using frequency spectrometry). However, they can also be measured using catalyzes and that is described below.

COF2 acts as catalyst and where it creates CS2 in cell lines and in tumors in vivo. CS2 also acts as catalyst and where it creates COF2 in cell lines and in tumors in vivo. Consequently, a small amount of COF2 acts as catalyst and creates CS2, which in its turn creates more COF2 and which in its turn creates more CS2. Hence, COF2 or CS2 generates a chain reaction that produces an increasing amount of COF2 and CS2. The molecule CS2 is toxic and the molecule COF2 is extremely toxic. It is proposed that long-term cell exposure to COF2 and CS2 may result in reduced apoptosis and uncontrolled cell growth, i.e. cancer.

COF2 and CS2 in a cell line or tumor also act as catalysts outside the cell line and outside the human body by means of their radiated energy [3] . A test tube was positioned outside the cell line or outside the body in the vicinity of the tumor. The test tube contained C (carbon), F (fluoride) and S (sulfur) dissolved in H2O (water). The radiated energy from COF2 acted as catalyst and created CS2 in the test tube. The radiated energy from CS2 acted as catalyst and created COF2. Catalyzes of COF2 and CS2 in a test tube can be used as a cancer marker and where the method is non-invasive. Alternatively the test tube may contain only C, F and H2O wherein only COF2 was created. Alternatively the test tube may contain only C, S and H2O wherein only CS2 was created. Catalyzes in a test tube can be used as a method to analyze presence of COF2 and CS2 in cell lines or tumors.

COF2 can be analyzed using FTIR (Fourier Transform Infrared Spectrometer), GC (Gas Chromatography) or GC/MS (Gas Chromatography-Mass Spectrometry). CS2 can be analyzed using GC or LC (Liquid Chromatography).

The following types of cancer have been analyzed and they all contain the molecules COF2 and CS2: brain cancer, breast cancer, colon cancer, larynx cancer, leukemia, liver cancer, lung cancer, lymphatic cancer, melanoma, pancreatic cancer, prostate cancer, squamous cell carcinoma, testicular cancer and uterine cancer.

The following cancer cell lines have been analyzed and they contain the molecules COF2 and CS2: cellosaurus cell line Mutu-3, lymphoblastic cell line LCL and epithelial cell line HONE-1.

3. Discussion

A novel model of cancer is presented. Cancer is created by a mechanism that contains two toxic molecules: COF2 and CS2. They have the ability to reproduce each other wherein a chain reaction occurs that produces vast amounts of COF2 and CS2. CS2 is toxic and COF2 is extremely toxic. It is proposed that they impair the immune system and where the result can be impaired apoptosis and uncontrolled cell growth, i.e. cancer.

Empiric results display that cancer cell lines and cancer tumors act as catalysts wherein they produce COF2 and CS2, also at a distance, i.e. in a test tube positioned outside the cell line or outside the body close to the tumor. Catalyzes of COF2 and CS2 can be used as cancer marker. The method can also be used in order to analyze content of cell lines and tumors, i.e. the cancer causing mechanism.

Reports display correlation between bacterial infections and cancer [5] [6] , between viral infections and cancer [7] [8] and between autoimmune diseases and cancer [9] [10] . Recent findings display that many pathogenic bacteria, virus and autoimmune diseases contain a generic part that consists of the molecules COF2 and CS2. These findings will be reported elsewhere. Cancer cannot occur on its own, i.e. without presence of COF2 and/or CS2. Presence of COF2 and/or CS2 is mandatory in order to start the chain reaction where the long-term result is production of vast amounts of COF2 and CS2. Many pathogenic bacteria and viruses as well as autoimmune diseases contain a generic mechanism that consists of the molecules COF2 and CS2. It is proposed that chronic bacterial, viral and autoimmune diseases may start catalyzes of COF2 and CS2 in the infected area. The result is, eventually, a chain reaction that produces vast amounts of COF2 and CS2 and, sometimes, subsequent cancer. Hence, bacterial, viral and autoimmune diseases can initiate a chain reaction where the result is cancer. They are not carcinogenic by their own.

Reports display that Fe2O3 in nanoparticles had a therapeutic effect on cancer cells in vitro and xenograft liver cancer in mice and where the result was increased inhibitory rate and ratio of apoptosis [11] . Another report describes the therapeutic mechanism of treating SMMC-7721 liver cancer cells using Fe2O3 nanoparticles. It was shown that it could cause cellular necrosis, induce cellular apoptosis and significantly inhibit cellular growth, all of which appeared to be dependent on the concentration of the Fe2O3 nanoparticles. Fe2O3 is the active substance in Ferumoxytol [12] . Stanford researchers discovered that Ferumoxytol triggered the immune system to destroy tumor cells [13] . Another study suggests that Fe2O3 nanoparticles in Ferumoxytol could be applied “off label” to protect the liver from metastatic seeds and potentiate macrophage-modulating cancer immunotherapies [14] .

The above studies display a correlation between Fe2O3 and therapeutic effect on cancer and in particular the therapeutic effect of Ferumoxytol. The following is proposed. Fe2O3 acts as a catalyst where it reduces (destroys) the molecule COF2 contained in cancer tumors and metastases. However, the molecule CS2 is not destroyed. Destruction of (only) COF2 inhibits further production of CS2 and which limits toxic exposure in cancer tumors and metastases and where the result is increased cellular necrosis and apoptosis. This type of catalyzes can be demonstrated in the following experiment. A test tube containing COF2 was positioned close to 10 grams of Fe2O3 during 15 minutes. Then the test tube was removed and its content of COF2 was analyzed; the result was zero content of COF2.

4. Conclusion

The overall conclusion is that it has been difficult to analyze the cause of cancer using state of the art technique. The present report displays a novel method. Molecules in cancer cell lines and tumors can be analyzed because they radiate energy that causes catalyzes outside the cell line and outside the body in a test tube wherein specific molecules are created (COF2 and CS2). These molecules can be analyzed using conventional measurement techniques such as FTIR, GC, GC-MS and LC. The conclusion is also that Fe2O3 reduces the molecule COF2 and hence, Fe2O3 may have an important role in cancer therapy.

Conflicts of Interest

The authors declare no conflicts of interest.

Cite this paper

W. Giertz, H. (2018) A Novel Model of Cancer. Journal of Cancer Therapy, 9, 383-387. doi: 10.4236/jct.2018.95034.

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