Lee, A., Qiao, Y., Grigoriev, G., Chen, J., Park-Min, K.H., Park, S.H., Ivashkiv, L.B. and Kalliolias, G.D. (2013) Tumor necrosis factor alpha induces sustained signaling and a prolonged and unremitting inflammatory response in rheumatoid arthritis synovial fibroblasts. Arthritis & Rheumatism, 65, 928-938.
http://dx.doi.org/10.1002/art.37853

has been cited by the following article:

• TITLE: Multifaceted role of TNF-α during the pathogenesis of rheumatoid arthritis

  AUTHORS: Ramanjaneya V. R. Mula, Rangaiah Shashidharamurthy

  KEYWORDS: Cytokines; Tumor Necrosis Factor-α; Rheumatoid Arthritis; Inflammation

  JOURNAL NAME: Advances in Bioscience and Biotechnology, Vol.4 No.10, October 16, 2013

  ABSTRACT: Tumor necrosis factor alpha (TNF-α) a cytokine has been shown to be the key player during the pathogenesis of several autoimmune inflammatory disorders (presumably sterile inflammation) including rheumatoid arthritis (RA). Several studies have shown that TNF-α is mainly involved in the proinflammatory responses. However recent studies have reported multifunctional role of TNF-α during the development of RA. Therefore, in this article we have highlighted the distinct functions of TNF-α during pathogenesis of RA.