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Pascual, R.M. and Peters, S.P. (2005) Airway remodeling contributes to the progressive loss of lung function in asthma: An overview. Journal of Allergy and Clinical Immunology, 116, 477-486. doi:10.1016/j.jaci.2005.07.011
has been cited by the following article:
TITLE: The airway epithelium dysfunction in the pathogenesis of asthma: The evidence
AUTHORS: Salvatore Leonardi, Alfina Coco, Michele Miraglia Del Giudice, Gianluigi L. Marseglia, Mario La Rosa
KEYWORDS: Airway Epithelium; Dysfunction; Asthma; Evidence
JOURNAL NAME: Health, Vol.5 No.2A, February 27, 2013
ABSTRACT: Asthma is traditionally defined as a chronic, multisystem, multicellular disease characterized by bronchial hyper-responsiveness (BHR) and lung inflammation. In this illness is particularly involving the airway epithelium as the place where the inflammation begins, develops and often returns through airway remodelling. In specific way there is also an evolving awareness of the active participation of structural elements, such as the airway epithelium, airway smooth muscle, and endothelium, in this process. The airway inflammation and remodelling together likely explain the clinical manifestations of asthma that is demonstrated by complicated respiratory manifestations in which wheeze occurring secondary to bronchoconstriction in the setting of airway hyper-responsiveness and mucous hypersecretion. This is confirmed by also the histopathological analyses of bronchial biopsies from asthmatic subjects. For this reason is very important to understand the physiological role of the airway epithelium and their changes during the exacerbation of disease.
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