Author(s)

Anders Ziegler¹, Magne Nødland², Dag Tveiten¹, Karl L. Reichelt^1,3*

¹Lab 1, No. 1337, Sandvika, Norway.
²Stavanger, Norway.
³University of Oslo, Oslo, Norway.

Problem: There is a need to collect the many and varied data on AD/HD (Attention deficit hyper-activity disorder) into a meaningful overview. Method: Based on peer reviewed and published data as well as own research we try to make sense of the physiological mechanisms resulting in the relevant symptoms. Conclusion: AD/HD clearly has a genetic disposition, but as with many other genetic syndromes, the resulting proteomics must be stressed to become manifest as disease/ disorder. A common trait in the different etiologies is lower arousal.

AD/HD, Arousal, Genetics, Peptides, Diet, PUFA (Poly Unsaturated Fatty Acids), Monamines

Ziegler, A. , Nødland, M. , Tveiten, D. and Reichelt, K. (2016) The AD/HD Syndrome as a Group of Biological Disorders. Open Journal of Pediatrics, 6, 22-28. doi: 10.4236/ojped.2016.61005.