"
Transient Receptor Potential Ion Channels in the Etiology and Pathomechanism of Chronic Fatigue Syndrome/Myalgic Encephalomyelitis"
written by D. Staines, S. Du Preez, H. Cabanas, C. Balinas, N. Eaton, R. Passmore, R. Maksoud, J. Redmayne, S. Marshall-Gradisnik,
published by
International Journal of Clinical Medicine,
Vol.9 No.5, 2018
has been cited by the following article(s):
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[1]
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Altered TRPM7-Dependent Calcium Influx in Natural Killer Cells of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome Patients
Biomolecules,
2023
DOI:10.3390/biom13071039
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[2]
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The Pathobiology of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome: The Case for Neuroglial Failure
Frontiers in Cellular Neuroscience,
2022
DOI:10.3389/fncel.2022.888232
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[3]
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Potential Implications of Mammalian Transient Receptor Potential Melastatin 7 in the Pathophysiology of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome: A Review
International Journal of Environmental Research and Public Health,
2021
DOI:10.3390/ijerph182010708
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[4]
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Current Research Provides Insight into the Biological Basis and Diagnostic Potential for Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS)
Diagnostics,
2019
DOI:10.3390/diagnostics9030073
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