[1]
|
Lacotte, S., Brun, S., Muller, S. and Dumortier, H. (2009) CXCR3, inflammation, and autoimmune diseases. Annals of the New York Academy of Sciences, 1173, 310-317.
doi:10.1111/j.1749-6632.2009.04813.x
|
[2]
|
Sherer, Y. and Shoenfeld, Y. (2006) Mechanisms of disease: Atherosclerosis in autoimmune diseases. Nature Clinical Practice Rheumatology, 2, 99-106.
doi:10.1038/ncprheum0092
|
[3]
|
Firestein, G.S. (2003) Evolving concepts of rheumatoid arthritis. Nature, 423, 356-361. doi:10.1038/nature01661
|
[4]
|
Gabriel, S.E. and Michaud, K. (2009) Epidemiological studies in incidence, prevalence, mortality, and comorbidity of the rheumatic diseases. Arthritis Research & Therapy, 11, 229. doi:10.1186/ar2669
|
[5]
|
Matsumoto, I., Staub, A., Banjoist, C. and Mathis D. (1999) Arthritis provoked by linked T and B cell recognition of a glycolytic enzyme. Science, 286, 1732-1735.
doi:10.1126/science.286.5445.1732
|
[6]
|
Schaller, M., Burton, D.R. and Ditzel, H.J. (2001) Autoantibodies to G6PI in rheumatoid arthritis: Linkage between an animal model and human disease. Nature Immunology, 2, 746-753. doi:10.1038/90696
|
[7]
|
Schaller, M., Stohl, W., Tan, S.M., Benoit, V.M., Hilbert, D.M. and Ditzel H.J. (2005) Raised levels of anti-glucose-6-phosphate isomerase IgG in serum and synovial fluid from patients with inflammatory arthritis. Annals of the Rheumatic Diseases, 64, 743-749.
doi:10.1136/ard.2004.025502
|
[8]
|
Schaller, M., Stohl, W., Benoit, V., Tan, S.M., Johansen, L. and Ditzel H.J. (2006) Patients with inflammatory arthritic diseases harbor elevated serum and synovial fluid levels of free and immune-complexed glucose-6-phosphate isomerase (G6PI). Biochemical and Biophysical Research Communications, 349, 838-845.
doi:10.1016/j.bbrc.2006.08.105
|
[9]
|
Fan, L.Y., Zong, M., Wang, Q., Yang, L., Sun, L.S., Ye, Q., Ding, Y.Y. and Ma, J.W. (2010) Diagnostic value of glucose-6-phosphate isomerase in rheumatoid arthritis. Clinica Chimica Acta, 411, 2049-2053.
doi:10.1016/j.cca.2010.08.043
|
[10]
|
Kassahn, D., Kolb, C., Solomon, S., Bochtler, P. and Illges, H. (2002) Few human autoimmune sera detect GPI. Nature Immunology, 3, 411-412.
doi:10.1038/ni0502-411b
|
[11]
|
Schubert, D., Schmidt, M., Zaiss, D., Jungblut, P.R. and Kamradt, T. (2002) Autoantibodies against GPI and creatine kinase in rheumatoid arthritis. Nature Immunology, 3, 411. doi:10.1038/ni0502-411a
|
[12]
|
Herve, C.A., Wait, R. and Venables, P.J. (2003) Glucose-6-phosphate isomerase is not a specific autoantigen in rheumatoid arthritis. Rheumatology, 8, 986-988.
doi:10.1093/rheumatology/keg271
|
[13]
|
Matsumoto, I., Lee, D.M., Goldbach-Mansky, R., Sumida, T., Hitchon, C.A., Schur, P.H., Anderson, R.J., Coblyn, J.S., Weinblatt, M.E., Brenner, M., Duclos, B., Pasquali, J.L., El-Gabalawy, H., Mathis, D. and Benoist, C. (2003) Low prevalence of antibodies to glucose-6-phosphate isomerase in patients with rheumatoid arthritis and a spectrum of other chronic autoimmune disorders. Arthritis & Rheumatism, 48, 944-954.
doi:10.1002/art.10898
|
[14]
|
Klippel, J.H., Crofford, L.J.C., Stone, J.H. and Weyand, C.M. (2001) Primer on the rheumatic diseases. 12th Edition, Arthritis Foundation, Atlanta.
|
[15]
|
Kraus, A.P., Langston Jr., M.F. and Lynch, B.L. (1968) Red cell phosphoglycerate kinase deficiency. A new cause of non-spherocytic hemolytic anemia. Biochemical and Biophysical Research Communications, 30, 173-177.
doi:10.1016/0006-291X(68)90466-X
|
[16]
|
Yu, F.L., Liao, M.H., Lee, J.W. and Shin W.L. (2004) Induction of hepatoma cells migration by phosphoglucose isomerase/autocrine motility factor through the upregulation of matrix metalloproteinase-3. Biochemical and Biophysical Research Communications, 314, 76-82.
doi:10.1016/j.bbrc.2003.12.056
|